Description
Nil
Fluorescence In Situ Hybridization (FISH) For MDS Panel (Cytogenetics + FISH (Del(5q), Del(7q), del(20q))
Nil
TAT:
Specimen Type:
Genomics Panel Test
Other Tests
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264%
NPM-1 Mutation Detection By PCR
Hotspot genes: ABL1, BRAF, CBL, CSF3R, DNMT3A, FLT3, GATA2, HRAS, IDH1, IDH2, JAK2, KIT, KRAS, MPL, MYD88, NPM1, NRAS, PTPN11, SETBP1, SF3B1, SRSF2, U2AF1, WT1. Full Genes: ASXL1, BCOR, CALR, CEBPA, ETV6, EZH2, IKZF1, NF1, PHF6, PRPF8, RB1, RUNX1, SH2B3, STAG2, TET2, TP53, ZRSR2. Fusion Driver Genes: ABL1, ALK, BCL2, BRAF, CCND1, CREBBP, EGFR, ETV6 (TEL), FGFR1, FGFR2, FUS, HMGA2, JAK2, KMT2A(MLL), MECOM, MET, MLLT10, MLLT3, MYBL1, MYH11, NTRK3, NUP214, PDGFRA, PDGFRB, RARA, RBM16, RUNX1 (AML1), TCF3(E2A), TFE3
NPM-1 Mutation Detection By PCR
Hotspot genes: ABL1, BRAF, CBL, CSF3R, DNMT3A, FLT3, GATA2, HRAS, IDH1, IDH2, JAK2, KIT, KRAS, MPL, MYD88, NPM1, NRAS, PTPN11, SETBP1, SF3B1, SRSF2, U2AF1, WT1. Full Genes: ASXL1, BCOR, CALR, CEBPA, ETV6, EZH2, IKZF1, NF1, PHF6, PRPF8, RB1, RUNX1, SH2B3, STAG2, TET2, TP53, ZRSR2. Fusion Driver Genes: ABL1, ALK, BCL2, BRAF, CCND1, CREBBP, EGFR, ETV6 (TEL), FGFR1, FGFR2, FUS, HMGA2, JAK2, KMT2A(MLL), MECOM, MET, MLLT10, MLLT3, MYBL1, MYH11, NTRK3, NUP214, PDGFRA, PDGFRB, RARA, RBM16, RUNX1 (AML1), TCF3(E2A), TFE3
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264%
OncQuer TM Germline Mutation Panel by NGS
Identification of gene mutations that increase risk of cancer or has therapeutic implications in an affected individual
OncQuer TM Germline Mutation Panel by NGS
Identification of gene mutations that increase risk of cancer or has therapeutic implications in an affected individual
-
264%
OncQuer TM Scout LB EGFR Resistance Test
Identification gene mutations leading to resistance to EGFR therapy
OncQuer TM Scout LB EGFR Resistance Test
Identification gene mutations leading to resistance to EGFR therapy
-
264%
OncQuer TM Scout LB EGFR Response Test
Identification gene mutations leading to sensitivity and resistance to EGFR therapy
OncQuer TM Scout LB EGFR Response Test
Identification gene mutations leading to sensitivity and resistance to EGFR therapy
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